Case study demonstrating best practice

By Amanda Palmer, Wound Management Consultancy.

This Case study won The National 3M Tegaderm Challenge 2003.

 

 

This case study presents the management of an elderly gentleman who had developed a pressure sore during a time of illness at home. It demonstrates how effective evidence based management as part of a team approach can effectively promote healing.

 

Pressure damage occurs rapidly as the pressure between the person’s skeleton and the object taking their weight compresses the capillaries within the tissues (Waterlow 1994). Joe was a 79-year-old gentleman who was admitted following a period of self-neglect due to the loss of his wife the previous year and a recent episode of influenza leading to a chest infection. He had mild congestive cardiac failure, was a non-smoker, with no significant previous medical history.

 

Over the previous 3 weeks he had spent much of his time sitting in his chair with his feet on a stool, feeling unwell. Joe lived alone, and his family lived in another town, so he had tried to manage alone and had eaten very little, become under-nourished, dehydrated and run down (Desai 1997).

 

The medical team requested a surgical review of the necrotic pressure sore on his right heel, and referral to myself to review the wound management. The surgical team felt that amputation might be necessary, but after discussion I negotiated with them to try dressings and pressure relief first and then review again in a weeks time to reassess progress.

 

Joe was referred to the dieticians to address his nutritional needs (Balance 2002), Social worker to review his home situation, support, and so forth. A dynamic pressure relief mattress and cushion were provided (Waterlow 1994).

 

 

Wound Assessment

 

Subjective:

Joe described having sat in his chair for most of the day and night over the previous couple of weeks. He noticed his heels were feeling sore just over a week ago, but hadn’t looked at them and hadn’t asked for any help. Joe described only a mild itch and numbness at this time, but no pain. I used a 1 – 10 pain scale to monitor his pain, 1 - no pain, to 10 - extreme pain (Allan 2002). Joe rated his heel as 2 at the initial assessment.

 

Objective:

The pressure sore wound was covered with a dehydrated, necrotic Escher of approximately 17cm squared. The edges were devitalised, with fluff and debris attached, as he had not realised the wound was present and so it had not been covered.

 

The peri-wound area was dry with mild erythema. The wound was not malodorous and there was no ooze or exudate at this initial assessment (Thomas 1997).

 

 

Plan

 

There were several priorities to managing this wound:

v    Re-hydrate the necrotic tissue

v    Prevent maceration

v    Control the bacterial loading

v    Encourage repair and regeneration of the capillaries – angiogenisis, and neoangiogenisis.

v    Prevent deterioration.

v    Be acceptable to Joe.

v    To be cost and time efficient.

 

In 1962 George Winter identified that a moist wound-healing environment was necessary for effective regeneration and migration of skin cells. His work has been supported and built upon over the past 4 decades. Since his initial research, wound product development has focused around achieving this environment. Turner (1982), Morrison and Moffat (1997) and Sussman and Bates-Jensen (1998) identify the aims of an ideal dressing:

v    Has thermal properties,

v    Manages exudate

v    Prevents further trauma,

v    Does not contaminate the wound

v    Protects the wound

v    Is comfortable and acceptable to the patient

 

With this in mind the first wound management plan consisted of mapping the wounds overall dimensions, and rehydrating. For this a hydrogel was applied, which was occluded with 3M Tegaderm. This was replaced at 24 hours and then at 48 hours. By this time the area was softening and the hydrogel was placed under the edges of the necrotic Escher as it was beginning to lift. The exudate levels were increasing as the local vascular system recovered (Sibbald 2002).

 

At day 5 I was able to remove 80% of the debriding Escher, to reveal fibrous sloughy tissue on the base of the wound (Sibbald 2002). This was hydrated and being broken down by the macrophages through the process of autolysis (Moore 1999). I estimated the wound as being full thickness, although this could still not be confirmed as too much devitalised tissue remained (Allan 2002). There were signs of infection, Joe was pyrexial at 38 degrees C, the wound exudate was increasing – although this was due in part to the rehydration, the peri-wound area has warm and erythema was present. There was an increase in the malodour from the wound, which Joe was finding difficult to tolerate. Broad-spectrum IV antibiotics had been commenced on admission due to the chest infection. As the wound bed was now accessible a swab was taken. The IV antibiotics were supported with topical application of a Cadexomer Iodine based product (Cooper and Lawrence 1996, Gilcrist 1997), covered with an absorbent pad and secured with 3M Tegaderm, to maintain the moist, warm wound environment which was proving so successful.

 

At the 7th day the surgical team returned to review progress. It was decided to continue providing the warm, moist environment and enabling the process of autolysis, that was proving effective, to continue. The pressure relief was significant in enabling neoangiogenisis, and the input of the dietician was helping to improve Joe’s nutritional status.

 

Joe was feeling better with the IV antibiotics and was mobilising daily with the physiotherapist, during this he wore an extra thick layer of padding over the pressure sore dressing. He had been able to talk to the chaplain service, and the social worker was looking into finding Joe a place in a retirement village. Joe was feeling generally more positive about the future.

 

The pressure sore continued to progress well with application of Cadexomer Iodine paste daily initially, and then alternate days from day 5 to day 11. By day 11 the wound had debrided, and the malodour was gone. Calcium alginate was commenced to fill the cavity and manage the exudate (Carville 1998), secured with 3M Tegaderm and left in situ for 3 days. The 3M Tegaderm enabled the nursing staff to monitor the calcium alginate and see when it had become a gel to know to change the dressing. By day 14 there was a healthy base of granulation tissue. The wound edges were contracting, leaving the wound now reduced to an area of 11cm squared. The exudate volumes were reducing, the malodour and erythema were gone. 3M Tegaderm remained pivotal in the dressing regimen, to secure the dressing and maintain the moist, warm environment. It was also comfortable for Joe and enabled him to mobilise gently and to shower without requiring re dressing.

 

Joe was discharged home with increased support and on the waiting list for his chosen retirement home. The district Nurses continued his wound management at home, and he began to socialise again.

 

This case study demonstrates how evidence based nursing management can prove so effective for patients. It also demonstrates how working as part of the multi disciplinary team can promote holistic healing for the client in a supportive, client centred way, if we consider that a dressing is only as good as the body it is placed on (MacLellan 1998). The attributes of 3M Tegaderm in providing a semi occlusive environment and the ability to monitor the wound without disturbing the dressing are significant in promoting client comfort and cost effectiveness.


References

 

 

Allan E et al “Wound assessment: Module 2” Monash Distance Education; Victorian College of pharmacy, Monash University, Australia. 2002

 

Balance K “Wound healing and nutrition” NZ Wound Care Society inc. Newsletter, Winter 2002, p4, New Zealand.

 

Carville K “General Assessment” in Wound Care Manual 3rd Ed., Silver Chain Foundation, Australia, 1998 pp. 36-42

 

Cooper and Lawrence J "The role of antimicrobial agents in wound care" Journal of Wound Care, vol 5, 1996, p 374 - 380.

 

Desai H "Aging and Wounds: Healing in old age" In Journal of wound care, vol 6, no. 5 May 1997, pp 237-239.

 

Gilchrist B "Should iodine be reconsidered in wound management" Journal of Wound Care, vol 6, no 3, 1997, p 148-150

 

Larocco M. (1994) “Inflammation and Immunity” IN Porth C.M (ed.) Pathophysiology. Concepts of Altered Health States. J.B. Lippincott Co. Philadelphia,

 

Maclellan L and Rice J “Modern Wound Management”, Pulse, no.2, 1998, 2 pages

 

Moore 1999 “Cell biology of chronic wounds: the role of inflammation” Journal of Wound Care, vol 8, no 7, July. P 345-348.

 

Morison M and Moffat C, Nursing Management of Chronic Wounds, 2nd Ed. Mosby, Spain, 1997.

 

Sibbold G “Wound Bed Preparation” Vascular conference paper, Rotorua, New Zealand, April 2002

 

Sussman C and Bates-Jensen B M Wound care: a collaborative practice manual for physical therapists and nurses. Aspin publishers, Maryland, 1998, pp. 21, 66-71, 73-82.

 

Thomas S “ Assessment and Management of Wound Exudate” Journal of Wound Care, vol 6, no 7, July 1997, p327–330

 

Turner T “Which dressing and why?” Nursing Times, vol 18, no 29, 1982, supp 1-3.

 

Waterlow J A 1994 “Pressure sore prevention Manual.” Newtons, England.

 

Winter G, 1962, Nature.